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Explore inspiring journeys of recovery and resilience as patients share their experiences with Padaav’s root-cause-focused, evidence-based Ayurvedic treatments, highlighting the power of personalized care and holistic healing.
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Delve into the heartfelt experiences of patients who have transformed their health and lives with Padaav’s research-backed Ayurvedic care, showcasing the impact of addressing root causes for lasting recovery.
Hamad Alam
Rectal Adenocarcinoma
New Delhi
Hamad’s struggle reached a terrifying peak in 2018 while traveling back to Delhi from a cou…
Shilipi
Acute Recurrent Pancreatitis
Kotdwar, Uttarakhand.
Shilpi’s struggle began suddenly during her 10th-grade school break, when she felt a sharp …
Anurag Goyal
Investment Banker
New Delhi
Anurag’s struggle began suddenly in the midnight of September 2010, when he experienced exc…
Sumit Kumar
Pancreas Divisum with secondary Chronic Pancreatitis
Dehradun, Uttarakhand
Sumit suffered from Pancreas Divisum, a congenital anomaly where the two pancreatic ducts (the ve…
Aviral Saxena
Chronic Pancreatitis with Secondary Diabetes
Dehradun, Uttarakhand
Aviral’s journey began in November 2022 while he was actively working as a freelancer. The …
Rajeev Sharma
Chronic Pancreatitis
Dehradun, Uttarakhand
Rajeev’s journey began with mild symptoms in 2009, potentially following a physical encount…
Research
Our Research
At Padaav, alongside delivering unparalleled patient care and promoting holistic health, we are committed to advancing Ayurveda by actively participating in research and contributing to its growing body of knowledge.
Impact Evaluation of Ayurvedic Treatment Protocol on Three HundredNineteen Cases of Different Variants of Pancreatitis
Pancreatitis, affecting the pancreas' function, causes symptoms like pain, nausea, and weight loss. Conventional treatments are limited, prompting many to seek alternative medicine. A North India Ayurvedic clinic successfully treated 319 cases of Recurrent Acute/Chronic Pancreatitis using a specialized Ayurvedic Treatment Protocol (ATP), including the formulation Amar. This treatment improved weight and reduced attack frequency without side effects. Ongoing research is exploring its potential.
Treatment of relapsed undifferentiated acute myeloid leukemia (AML-M0) with Ayurvedic therapy
A 16-year-old boy with AML-M0 relapsed after chemotherapy in 1997. He opted for Ayurvedic therapy (AYT) instead of further treatment. After six months, he was in remission and continued AYT for five years, with intermittent maintenance for two years. He has been disease-free and healthy for 12 years.
Randomized Controlled Study to Evaluate Prophylactic Properties of Ayurvedic Treatment Protocol in Refractory and Chronic Migraine Patients
This study compared Ayurvedic treatment to conventional therapy for chronic refractory migraine. After 360 days, the Ayurvedic group showed greater reductions in headache days, VAS, and MIDAS scores (p
Scientific validated approach for application of Mandura bhasma A review
Mandura (iron rust) is purified and calcined in Ayurveda to create Mandura Bhasma, used for conditions like spleen and liver enlargement, jaundice, oedema, and anemia. Our study found that Prak-20 aligns with the classical dose schedule for Mandura Bhasma.
Significant Reduction of Carbohydrate 19-9 Antigen Levels in a Patient of Acute on Chronic Pancreatitis through Ayurvedic Treatment Protocol
A 52-year-old male with recurrent acute necrotizing pancreatitis showed poor response to conventional treatment, leading to weight loss and high CA 19-9 levels. Ayurvedic therapy, including herbal formulations, a balanced diet, and rest, normalized his CA 19-9 levels within 108 days, highlighting its potential for further study.
Clinical Intervention of Ayurvedic Treatment in the Patients of Recurrent Acute/Chronic Pancreatitis
The study of 1233 acute pancreatitis patients found that 12.9% experienced oral feeding intolerance (OFI). Risk factors included younger age, male sex, alcohol use, smoking, non-biliary etiology, and SIRS at 48h. SIRS at 48h (OR 2.85) and non-biliary etiology (OR 1.77) were independently linked to OFI, with biliary etiology being protective.