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Explore inspiring journeys of recovery and resilience as patients share their experiences with Padaav’s root-cause-focused, evidence-based Ayurvedic treatments, highlighting the power of personalized care and holistic healing.
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Delve into the heartfelt experiences of patients who have transformed their health and lives with Padaav’s research-backed Ayurvedic care, showcasing the impact of addressing root causes for lasting recovery.
Hamad Alam
Rectal Adenocarcinoma
New Delhi
Hamad’s struggle reached a terrifying peak in 2018 while traveling back to Delhi from a cou…
Patient Name
Acute Recurrent Pancreatitis
Kotdwar, Uttarakhand.
Shilpi’s struggle began suddenly during her 10th-grade school break, when she felt a sharp …
Anurag Goyal
Investment Banker
New Delhi
Anurag’s struggle began suddenly in the midnight of September 2010, when he experienced exc…
Sumit Kumar
Pancreas Divisum with secondary Chronic Pancreatitis
Dehradun, Uttarakhand
Sumit suffered from Pancreas Divisum, a congenital anomaly where the two pancreatic ducts (the ve…
Aviral Saxena
Chronic Pancreatitis with Secondary Diabetes
Dehradun, Uttarakhand
Aviral’s journey began in November 2022 while he was actively working as a freelancer. The …
Rajeev Sharma
Chronic Pancreatitis
Dehradun, Uttarakhand
Rajeev’s journey began with mild symptoms in 2009, potentially following a physical encount…
Research
Our Research
At Padaav, alongside delivering unparalleled patient care and promoting holistic health, we are committed to advancing Ayurveda by actively participating in research and contributing to its growing body of knowledge.
Randomized Controlled Study to Evaluate Prophylactic Properties of Ayurvedic Treatment Protocol in Refractory and Chronic Migraine Patients
This study compared Ayurvedic treatment to conventional therapy for chronic refractory migraine. After 360 days, the Ayurvedic group showed greater reductions in headache days, VAS, and MIDAS scores (p
Significant Reduction of Carbohydrate 19-9 Antigen Levels in a Patient of Acute on Chronic Pancreatitis through Ayurvedic Treatment Protocol
A 52-year-old male with recurrent acute necrotizing pancreatitis showed poor response to conventional treatment, leading to weight loss and high CA 19-9 levels. Ayurvedic therapy, including herbal formulations, a balanced diet, and rest, normalized his CA 19-9 levels within 108 days, highlighting its potential for further study.
Remission in a Relapse Case of Acute Promyelocytic Leukaemia for Twenty-two years using Metal Based Ayurvedic Treatment: A Case Report
Acute Promyelocytic Leukemia (APML) is a blood cancer with symptoms like anemia, fatigue, and easy bleeding. While modern treatments improve outcomes, relapses and side effects persist. This case report discusses an APML patient who, after relapse, opted for Ayurvedic treatment with metal-based formulations, achieving remission within weeks. The patient has remained disease-free for 22 years.
Hepato-protective Effect of an Ayurvedic Formulation Prak-20 in CCl 4 Induced Toxicity in Rats: Results of Three Studies
Prak-20, an ayurvedic herbo-mineral formulation, was studied for its hepato-protective effects in rats. It showed no significant liver damage or elevated enzyme levels compared to controls. In higher doses, it reduced liver enzymes by up to 54% and minimized liver damage. These results suggest Prak-20 is effective in protecting the liver, with further research needed on its mechanism.
Immunomodulatory Effects of a Herbo-Mineral Ayurvedic Formulation in Experimental Models
IMMBO, a herbo-mineral Ayurvedic formulation, shows promise as an immune-modulator in immunosuppressed rats. It enhances immunity, blocks histamine release, and modulates key molecules like TNF-α and NF-κB, offering potential for allergic and immune-related disorders.
Clinical Intervention of Ayurvedic Treatment in the Patients of Recurrent Acute/Chronic Pancreatitis
The study of 1233 acute pancreatitis patients found that 12.9% experienced oral feeding intolerance (OFI). Risk factors included younger age, male sex, alcohol use, smoking, non-biliary etiology, and SIRS at 48h. SIRS at 48h (OR 2.85) and non-biliary etiology (OR 1.77) were independently linked to OFI, with biliary etiology being protective.